Respiratory syncytial virus infection in guinea pig β-adrenergic receptor
, B receptor function is normal in the initial infection , with the duration of the continuation and intensification of the disease gradually decreased ,
moncler jassen, Kd values change and the extent of pathological changes in lung tissue is consistent . The results suggest that decreased B receptor affinity is not the inherent characteristics of infected individuals , but by RSV infection in the body accompanied by the emergence of a regulatory change is the result of secondary changes . Riedel , etc. ] · 1 to RSV infection in guinea pigs and the control group were injected with the same dose of acetylcholine ,
######## oakley sunglasses, by volume were measured by scanning keiki two changes in airway resistance in guinea pigs showed that RSV infection of airway resistance decreased level was significantly higher. Bramley also used the same method , etc. J reached the same conclusion. Experiments confirmed the above , RSV infection of airway response was significantly increased . RSV in addition to a direct result of airway mucosal injury , but also as an allergen causing the allergic airway inflammation . An imbalance of nerve receptors in addition to the mechanism involved , such as the B receptor function, cholinergic hyperactivity 4J, weakening the role of non- adrenergic [ 5l, non-cholinergic nerves increased the release of sensory neuropeptides [ J et al . An imbalance of nerve receptors in the mechanism ,
uggs nederland, B receptor function changes on the changes in airway responsiveness is especially important to stimulate B receptor can lead to decreased microvascular permeability ,
abercrombie bruxelles, ciliary movement increased, increased release of mast cells and other inflammatory mediators . In this experiment ,
canada goose jakke, RSV infection in guinea pigs , the lung tissue was significantly reduced receptor function p , p -receptor-mediated biological effects of these also will be reduced , so that was a guinea pig airway smooth muscle slow loss of status , vascular permeability increase in mucus hypersecretion , leading to a guinea pig airway hyperresponsiveness generation. [
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